Aftermath of Haiti Earthquake: Dust Risks of Cement RubbleHomeopathy:Silica terra is the first remedy we think of to address the issue of foreign matter or foreign particles in the system, whether it be a splinter or stone embedded in the skin or dust inhaled in the lungs. PubMed has an article by Dr Guermonprez for silicosis and the homeopathic remedy silica. Silica is a mineral and is prepared from silicon dioxide found in flint, quartz, sandstone and other common rocks. Polthos foetidus is an asthma remedy specifically worse from dust inhalation. Skunk cabbage is found in the pharmacopoeia as "dracontium" for respiratory diseases, nervous disorders, rheumatism and dropsy. http://en.wikipedia.org/wiki/Eastern_Skunk_CabbageKali bichromate (potassium bichromate) helps the typical sinusitis with hard yellow mucus. Baryta carb. Could this address the issues that barium carbonate toxicity would present? Nasal Irrigation: Other treatments include nasal irrigation with the neti pot to loosen and clean the nasal passages. Silicosis: Silica dust gets trapped in the tiny alveoli sacs of the lungs. Three Stages of Silicosis: During the first stage, the patient feels some difficulty in breathing, has a mild cough and sputum production. During the second stage, the patient also complains of chest pain and reduced work capacity. During the third stage, the disease becomes life threatening. The patient becomes almost helpless, and symptoms like severe cough, bloodstained sputum, continuous chest pains and rapid weight loss become very prominent, eventually leading to premature death. Without these signs, silicosis can be difficult to diagnose; nodules that collect to form a mass can take up to 20 years to be identified in chest x-rays. The victim thus notices the symptoms only after several years of exposure. By Deepak Malik. http://boloji.com/health/articles/01045.htm Reference: P. 199 The Family Guide to Homeopathy: Symptoms and Natural Solutions by Andrew Lockie Squilla: Dry, staccato cough, sneezing, stitching pain in chest, wanting to take deep breaths Aconite: Sharp pain comes on suddenly after exposure to cold dry wind, breathlessness, anxiety, fear of dying Cantharis: Fluid on lung causes breathlessness and burning pains, mild fever, heartbeat rapid and irregular, frequent dry cough Belladonna: Pain comes on very suddenly, face flushed and hot, great thirst, delirium Hepar sulph: When recovery from pleurisy is slow, and complicated by fluid on lung
>Rubble Dust: A Health Risk For Haiti's Earthquake Survivors?http://www.npr.org/blogs/health/2010/01/rubble_dust_a_health_risk_f... It's one of the lasting images of January 12 -- the enormous dust cloud hanging over Port-au-Prince minutes after Haiti's massive earthquake. And then there were all those survivors' faces, coated with thick white masks of rubble dust. Today, the images from Haiti show countless numbers of people sleeping in the streets, breathing dust-laden air, day and night. All these images have triggered worry among health experts who have monitored first responders caught in the dust cloud when the World Trade Center towers collapsed on Sept. 11. They wonder, will Haitian earthquake survivors suffer the same kinds of lung problems?
"There's probably some reason to worry," says Dr. Philip Landrigan, chairman of preventive medicine at Mount Sinai Medical Center in New York. "It may be there are people caught in that dust cloud who suffered exposures similar to those at the World Trade Center. It's quite possible."
On 9/11 and its aftermath, more than 15,000 New York City firefighters, officers, EMTs and fire department retirees took part in rescue, recovery and fire suppression efforts. Long-term studies have shown that two-thirds have new or worsened respiratory problems years later.
"The actual effect of the dust and debris that rained down on our workforce on 9/11 may not be evident for years to come," says Dr. David Prezant, medical director of the New York Fire Department.
Nobody has collected any data on the kinds of exposures the Haiti earthquake survivors have experienced. Privately, federal health officials say they're frustrated at not being able to collect dust and air samples and monitor the population for respiratory problems. So the risk assessment so far has a matter of guesswork and speculation.
There are important similarities and differences between the likely hazards in Haiti's earthquake zone and at the World Trade Center site.
Landrigan of Mount Sinai says one similarity is that a major component of the dust in both places is pulverized concrete. "After 9/11, it comprised two-thirds of the dust -- the biggest single component," he says. "That's significant because powdered concrete tends to be very, very alkaline -- very caustic. So it's very irritating to the nasal passages, the throat and down into the lung."
One difference is probably fortunate for Haiti. "There just wasn't the amount of kinetic energy there as with the collapse of two 100-story towers," Landrigan says. "In New York, you had very, very high concentrations of dust. As a result of the force, the dust particles were very small."
The combination of small particle size and enormous concentrations of dust meant that when anyone caught in the World Trade Center dust cloud inhaled, Landrigan says, "there was so much stuff in the air that it overcame the normal defenses and the toxic dust went straight into their lungs. That normally doesn't happen."
On the other hand, the number of people exposed to dust from the earthquake rubble was far larger than in lower Manhattan on Sept 11. And many breathing the earthquake rubble dust are children, elderly people and those with pre-existing health problems.
Also, their exposure will occur over a much longer time.
The earthquake dust may not be washed out of the air and off the streets until Haiti's rainy season begins in April. Public health specialists say that will bring other health problems to the hundreds of thousands of homeless survivors.
Pneumoconiosis: Literally, “dust in the lungs,” causing breathlessness and coughing. Condition is most common among coal miners, though it may take up to 25 years to develop; inhaled particles of coal settle in lung tissue, causing patches of irritation that scar as they heal; if irritation and scarring become extensive, efficiency of lungs is drastically reduced.
Silicosis, a related dust disease that affects quarry workers, and metal grinder, etc., and may take only 10 years to develop; Asbestosis may take only 5 years. Silicosis brings with it increased risk of Tuberculosis, and asbestosis a high risk of Cancer of the Lung. Silicosis occurs when silica dust gets trapped in the tiny alveoli sacs in the lungs.
Coal, silica, aluminum, iron, beryllium, talc, asbestos, and synthetic fibers can all cause lung damage if inhaled daily for long periods. Since damage is irreversible, purpose of both orthodox and homeopathic treatment is to prevent complications.
There is no specific treatment for silicosis. Removing the source of silica exposure is important to prevent the disease from getting worse. Supportive treatment includes cough medicine, bronchodilators, and oxygen if needed. Antibiotics are prescribed for respiratory infections as needed.
Treatment also includes limiting exposure to irritants, quitting smoking, and having routine tuberculosis skin tests.
Thousands of construction workers are exposed to concrete every day without harm. But anyone who uses or supervises the use of portland cement should know its health hazards and the safe working procedures necessary to minimize exposure. This article outlines those hazards and makes recommendations on how to use cement safely.
Health effects Cement can cause ill health by skin contact, eye contact, or inhalation. Risk of injury depends on duration and level of exposure and individual sensitivity. Hazardous materials in wet concrete and mortar include: • alkaline compounds such as lime (calcium oxide) that are corrosive to human tissue • trace amounts of crystalline silica which is abrasive to the skin and can damage lungs • trace amounts of chromium that can cause allergic reactions.
Skin contact The hazards of wet cement are due to its caustic, abrasive, and drying properties.
Wet concrete contacting the skin for a short period and then thoroughly washed off causes little irritation. But continuous contact between skin and wet concrete allows alkaline compounds to penetrate and burn the skin.
When wet concrete or mortar is trapped against the skin—for instance, by falling inside a worker’s boots or gloves or by soaking through protective clothing—the result may be first, second, or third degree burns or skin ulcers. These injuries can take several months to heal and may involve hospitalization and skin grafts.
Ironically, severe cases often occur when personal protective clothing or equipment is worn. Wet concrete may get trapped inside rubber boots or gloves or gradually soak through coveralls. Concrete finishers kneeling on fresh concrete have had their knees severely burned. Corrosive bleed water from the concrete is absorbed by the worker’s pants and held against the skin for prolonged periods.
Cement dust released during bag dumping or concrete cutting can also irritate the skin. Moisture from sweat or wet clothing reacts with the cement dust to form a caustic solution.
Allergic skin reaction
Some workers become allergic to the hexavalent chromium in cement. A small yet significant percentage of all workers using cement will develop an allergy to chromium, with symptoms ranging from a mild rash to severe skin ulcers.
In addition to skin reactions, hexavalent chromium can cause a respiratory allergy called occupational asthma. Symptoms include wheezing and difficulty breathing. Workers may develop both skin and respiratory allergies to hexavalent chromium.
It’s possible to work with cement for years without any allergic skin reaction and then to suddenly develop such a reaction. The condition gets worse until exposure to even minute quantities triggers a severe reaction. The allergy usually lasts a lifetime and prevents any future work with wet concrete or powder cement.
Exposure to airborne dust may cause immediate or delayed irritation of the eyes. Depending on the level of exposure, effects may range from redness to chemical burns and blindness.
Inhaling high levels of dust may occur when workers empty bags of cement. In the short term, such exposure irritates the nose and throat and causes choking and difficult breathing. Sanding, grinding, or cutting concrete can also release large amounts of dust containing high levels of crystalline silica. Prolonged or repeated exposure can lead to a disabling and often fatal lung disease called silicosis. Some studies also indicate a link between crystalline silica exposure and lung cancer.
PubMed: http://www.ncbi.nlm.nih.gov/pubmed/15448758 Saudi Med J. 2004 Sep;25(9):1153-9. Health hazards of cement dust. Meo SA. Department of Physiology (29), College of Medicine, King Khalid University, PO Box 2925, Riyadh 11461, Kingdom of Saudi Arabia. firstname.lastname@example.org Even in the 21st century, millions of people are working daily in a dusty environment. They are exposed to different types of health hazards such as fume, gases and dust, which are risk factors in developing occupational disease. Cement industry is involved in the development of structure of this advanced and modern world but generates dust during its production. Cement dust causes lung function impairment, chronic obstructive lung disease, restrictive lung disease, pneumoconiosis and carcinoma of the lungs, stomach and colon. Other studies have shown that cement dust may enter into the systemic circulation and thereby reach the essentially all the organs of body and affects the different tissues including heart, liver, spleen, bone, muscles and hairs and ultimately affecting their micro-structure and physiological performance. Most of the studies have been previously attempted to evaluate the effects of cement dust exposure on the basis of spirometry or radiology, or both. However, collective effort describing the general effects of cement dust on different organ and systems in humans or animals, or both has not been published. Therefore, the aim of this review is to gather the potential toxic effects of cement dust and to minimize the health risks in cement mill workers by providing them with information regarding the hazards of cement dust. Barium Carbonatehttp://www.lycos.com/info/barium--barium-carbonate.html
Barium carbonate dust has been reported to be a bronchial irritant. Barium oxide dust is considered a dermal and nasal irritant (Beliles 1994). The effect of barium dusts on welders was investigated under simulated working conditions over a one-week time period (Zschiesche et al. 1992). Barium fume concentrations were 4.4 and 2.0 mg/m3 during welding with stick electrodes and flux cored wires, respectively. No adverse health effects on the welders were attributable to barium exposure, but there was a slight decrease in plasma potassium levels at the end of the work shift.
Toxic barium levels may be an origin of Multiple Sclerosis. According to some very recent research, high levels of industrial sources of barium (Ba) have been associated with a high incidence of Multiple Sclerosis.>
Some of the most toxic Multiple Sclerosis (MS) clusters of barium have been found in Saskatchewan, Sardinia, Massachusetts, Colorado, Guam, and NE Scotland where elevated levels of barium have been found in the soil. A toxic mean level of 1428 ppm of barium was compared to a non-toxic mean level of 19 ppm of barium recorded in adjoining MS-free regions. Sources of Barium: 1. Barium ore quarries 2. Heavy additives for oil-well-drilling mud
3. Atmospheric aerosol sprays for enhancing/refracting the signaling of radio/radar waves along military jet flight paths (including chemtrails) and missile test ranges
4. Paper and rubber industries
5. Fillers or extenders in cloth, ink, and plastics products
6. Radiography - the "barium milkshake"
7. Getter (scavenger) alloys in vacuum tubes
8. Deoxidizers for copper
9. Lubricants for anode rotors in X-ray tubes
10. Spark-plug alloys
11. Expensive white pigments In laymen's terms, this is how toxic barium levels connect to a diagnosis of MS: abnormal levels of barium salts can initiate the formation of MS as a result of a combination of barium with sulphate.
This in turn deprives sulphated molecules of their sulphate co-partner, and this disrupts the fibroblast growth factor, which induces the loss of S-proteoglycan activity, which maintains the growth and structural integrity of the myelin sheath. Whew.
Barium toxicity also disturbs the sodium-potassium ion pump capability -- another key feature in an MS profile. Ms could pivot upon a common disruption of the sulphate signaling systems in the body. Toxic levels of barium appear to disrupt this signaling system. Most of the health risks are caused by breathing in air that contains barium sulphate or barium carbonate. Barium is a silvery-white metal that can be found naturally in the environment. It is typically found combined with other chemicals, such as sulfur, carbon or oxygen. It is very light element, with a density half that of iron. Barium oxidizes in the air and reacts vigorously with water, liberating hydrogen. Barium reacts with almost all the non-metals, often forming poisonous compounds.
Barium is surprisingly abundant in the Earth's crust, and is the 14th most abundant element. High amounts of barium are only found in soils and in food, such as nuts, seaweed, fish and plants. Because of the extensive use of barium in industry, it is commonly released into the environment. As a result, barium concentrations in the air, water and soil are higher than naturally occurring concentrations.
Barium enters the air during mining processes, refining processes, the spraying of chemtrails, and during the production of barium compounds. It can also enter the air during coal and oil combustion.
Many hazardous waste sites contain barium. People that live near these sites can be exposed to harmful levels, and exposure can be by breathing dust, eating from the soil or plants grown in the soil, or by drinking water that has become polluted with barium.
Barium compounds that dissolve in drinking water can be harmful to human health. Toxic amounts of barium may cause paralyses and in some cases, death.
Small amounts of water-soluble barium can cause a person to experience breathing difficulties, increased blood pressures, heart rhythm changes, stomach irritation, muscle weakness, changes in nerve reflexes, swelling of the brain and liver, kidney and heart damage. And now, MS. Read more about Chronic barium intoxication and Multiple Sclerosis.
Homeopathic Educational Services: Prisma: The Arana of Materia Medica Illuminated ~ Similars and Parallels Between Substance and Remedy by Frans Vermeulen http://www.homeopathic.com/store/product=1194 Barium Carbonate. Witherite. CLASSIFICATION Barium, from Gr. barys, heavy, was distinguished from lime by the Swedish chemist Karl Scheele in 1774. Lavoisier included baryte or barote in 1789 in his list of simple substances as Terre pesante [heavy earth]. The English scientist Sir Humphrey Davy first isolated the element in 1808. It is found only in combination with other elements, mainly with sulphate [barite] and carbonate [witherite]. It belongs to group 2 [formerly group IIA] of the periodic table, called the alkaline earth group, and resembles calcium chemically. It is a soft metal with a silvery white lustre [like lead] when pure. Because it very easily oxidizes [turning black or greyish] it should be kept under petroleum or other oxygen-free liquids to exclude air. Water or alcohol decomposes barium. Barium minerals are dense, but barium itself is comparatively light. USES Alloys of barium with aluminium [or magnesium] are used as getters in electron tubes [e.g. in television sets, computer monitors, X-ray tubes], where they perfect the vacuum by combining with the last traces of various gases, and thus prolong the life of the device.
The screen of audio-visual equipment [television, computer, etc.] contains 4 to 10% barium carbonate to absorb roentgen rays arising in the electron tube. To achieve the same effect in colour televisions and colour [computer] monitors, the barium is combined with the even more active strontium. Barium protects by immobilizing and shielding. An alloy of barium with nickel is used in spark plugs and engine rod bearings.
The carbonate is used in ceramics, paints, enamels, marble substitutes, rubber; for the manufacture of paper, barium salts, electrodes, optical glasses; as an analytical reagent; and as a rat poison and insecticide. Barite [barium sulphate] is used as an X-ray contrast medium [barium meal]; as a weighing agent in oilwell drilling fluids; for the manufacture of photographic papers, artificial ivory, cellophane; as a filler for rubber, linoleum, oil cloth, polymeric fibres and resins, lithographic inks; as a water-colour pigment for coloured paper, in wallpaper; as a size for modifying the colours of other pigments; in heavy concrete for radiation shield.1 Barium powder is highly inflammable and may ignite spontaneously in air [at room temperature]. Various barium salts are employed in pyrotechnics [imparting a bright yellowish green colour to fireworks], signal flares [green], explosives, rocket propellant, matches, or as fireproofing agents. EFFECTS
Barium carbonate is almost insoluble in water. The compound is poisonous and may cause, in case of overexposure: salivation, vomiting, severe abdominal pain, violent diarrhoea; increased blood pressure; tinnitus, giddiness; muscle twitching, convulsions, paralysis; dilated pupils; confusion, somnolence; cardiac arrest; death due to respiratory failure. Acute poisoning is characterized by muscle weakness, followed by paralysis of the limbs and lungs, with additional heart problems. Victims usually remain conscious. Long term inhalation of barium dust may lead to deposition in the lungs, producing a form of pneumoconiosis called baritosis. Pneumoconiosis, or anthracotic tuberculosis, is characterized by pain in the chest, cough with little or no expectoration, despond, reduced thoracic excursion, sometimes cyanosis, and fatigue after slight exertion.2 Fibrogenic forms of pneumoconiosis include silicosis [caused by silica dust], asbestosis, anthracosis [caused by coal dust], and berylliosis [caused by beryllium dust or fumes]. Nonfibrogenic forms are considered to be benign and include siderosis [caused by iron dust], stannosis [caused by tin dust], and baritosis.
Dr Wilson of London has lately described a distinct case of poisoning with the carbonate. The quantity taken was half a teacupful; but emetics were given, and operated before any symptoms showed themselves. In two hours the patient complained of dimness of sight, double vision, headache, tinnitus, and a sense of distension in the stomach, and subsequently of pains in the knees and cramps of the legs, with occasional vomiting and purging next day; for some days afterwards the head symptoms continued, though more mildly, and she was much subject to severe palpitations. Mr Parkes mentions that, according to information communicated to him by the proprietor of an estate in Lancashire, where carbonate of baryta abounds, many domestic animals on his estate died in consequence of licking the dust of the carbonate, and that it once proved to two persons, a woman and her child, who took each about a dram. Dr Johnstone says he once swallowed ten grains of this compound, without experiencing any bad effect.
Experimentally, retarded growth can be evoked in rats and guinea pigs by placing them on a barium-free diet. The same effect occurs with a strontium-free diet. Blood pressure increases significantly in rats exposed to 10 to 100 ppm barium in their drinking water. In humans there appears to be increased risk of cardiovascular disease if the drinking water contains barium levels of 1.1 to 10 mg per litre. Both barium and strontium belong to the alkaline earth group, which comprises furthermore beryllium, magnesium, calcium, and radium. Magnesium and calcium are important physiologic elements; strontium and barium are closely related to calcium, but are not known to have any physiologic function and thus are foreign to the body.
As a foreign substance barium produces poisonous and untoward effects much easier than the physiologic calcium. In experiments many similarities are shown to the action of excessive doses of calcium. The anti-swelling, solidifying and therefore slowing effect on exchange and function of calcium action is much greater in barium and leads sooner to persistent states of pathology.
4 Soluble salts of barium are highly toxic and are used to control wide animals such as wolf, bear, etc.
ENVIRONMENT Barium gets into the air during the mining, refining and production of barium compounds, and from the burning of coal and oil. Barium compounds that dissolve easily are found in lakes, rivers, and streams. Fish and aquatic organisms accumulate barium. Most soils contain low levels of barium. Barium entering the body through the lungs seems to enter the bloodstream very easily, while it doesn't seem to enter the bloodstream as well from the stomach or intestines.
http://boloji.com/health/articles/01045.htmSilicosis develops over time when dust-containing silica is inhaled repeatedly into the lungs. Large amounts of organic dust particles (their sizes vary from 0.1 micron to 150 microns) exist in mines, and smaller particles (smaller than 5 microns) may become airborne and respirable. When the dust of the mines is inhaled, silica particles end up in the air sacs of the lung. The body's defence system quickly attacks the silica particles, releasing enzymes and radicals. The release of these by-products can result in death of lung tissues and white blood cells, which causes inflammation. The inflammation and scarring that damages the lung sacs prevents gas exchange and normal breathing. Damage to the lung tissue means that the capacity of the lungs to supply oxygen to the blood is reduced. The disease progresses in three stages. During the first stage, the patient feels some difficulty in breathing, has a mild cough and sputum production. During the second stage, in addition the patient complains of chest pain and reduced work capacity. During the third stage, the disease becomes life threatening. The patient becomes almost helpless, and symptoms like severe cough, bloodstained sputum, continuous chest pains and rapid weight loss become very prominent, eventually leading to premature death. Without these signs, silicosis can be difficult to diagnose; nodules that collect to form a mass can take up to 20 years to be identified in chest x-rays. The victim thus notices the symptoms only after several years of exposure, nor is there treatment for the disease only diagnosed.
Deepak Malik recounts the history of the mineworkers' plight.
Silicosis - Ayurvedic Herbal Treatmenthttp://ezinearticles.com/?Silicosis---Ayurvedic-Herbal-Treatment&am... Silicosis is a fibro-nodular lung disease caused by inhalation of dust containing crystalline silica, which is present in granite, slate and sandstone. Silicosis can be either acute or chronic. Common symptoms include cough, breathlessness, wheezing, digital clubbing, cyanosis, and other symptoms of cor pulmonale. This condition is an occupational hazard for people involved with mining, quarrying, sand blasting, cement and glass manufacturing, masonry, blast furnaces, coal mining and construction work.
The Ayurvedic treatment of silicosis is aimed at treating symptoms and preventing long term complications like chronic bronchitis, cor pulmonale, respiratory failure and lung cancer. Medicines like Shwas-Kuthar-Ras, Sitopaladi-Churna, Talisadi-Churna, Kantakari-Avaleha, Vasa-Avaleha, Ras-Sindur, Malla-Sindur, Sameer-Pannag-Ras and Shrung-Bhasma can be used to reduce inflammation in the lungs, and symptoms like cough, breathlessness and wheezing. Herbal medicines which can be used for this purpose include Pippali (Piper longum), Yashtimadhuk (Glycerrhiza glabra), Kantakari (Solanum xanthocarpum), Bruhat-Kantakari (Solanum indicum), Som (Ephedra vulgaris), Karkatashrungi (Pistichia integerima), Kulingan (Alpimia galangal), Dhatura (Dhatura fastuosa), Vasa (Adhatoda vasaka), Kushtha (Saussurea lappa) and Pushkarmool (Inula racemosa).
Medicines like Suvarna-Malini-Vasant, Suvarna-Bhasma, Abhrak-Bhasma, Maha-Laxmi-Vilas-Ras and Suvarna-Sameer-Pannag-Ras can be used to prevent permanent damage to the lungs and chronic complications like cor pulmonale and lung cancer. Medicines like Shatavari (Asparagus racemosus), Ashwagandha (Withania somnifera), Tulsi (Ocimum sanctum), Bhrungraj (Eclipta alba), Bala (Sida cordifolia) and Naagbala (Grewia hirsuta) can be used to boost the immune status of the body. Yashtimadhuk and Pippali, boiled in milk, are given in progressively increasing doses, followed by gradually tapering doses. Repeated such courses are very useful in preventing recurrent infections in the lungs, and help in repairing the damage done to the lungs.
Silica and Cancerhttp://www.lookchem.com/SILICA-CRYSTALLINE-ndash-TRIDYMITE/ Consensus of Silica , Cristobalite&ndash ; Tridymite (15468-32-3): NTP 10th Report on Carcinogens. IARC Cancer Review: Group 2A IMEMDT IARC Monographs on the Evaluation of Carcinogenic Risk of Chemicals to Man . 7 ,1987,p. 341.(World Health Organization, Internation Agency for Research on Cancer,Lyon, France.: ) (Single copies can be ordered from WHO Publications Centre U.S.A., 49 Sheridan Avenue, Albany, NY 12210) ; Animal Sufficient Evidence IMEMDT IARC Monographs on the Evaluation of Carcinogenic Risk of Chemicals to Man . 42 ,1988,p. 209.(World Health Organization, Internation Agency for Research on Cancer,Lyon, France.: ) (Single copies can be ordered from WHO Publications Centre U.S.A., 49 Sheridan Avenue, Albany, NY 12210) ; Human Limited Evidence IMEMDT IARC Monographs on the Evaluation of Carcinogenic Risk of Chemicals to Man . 42 ,1988,p. 209.(World Health Organization, Internation Agency for Research on Cancer,Lyon, France.: ) (Single copies can be ordered from WHO Publications Centre U.S.A., 49 Sheridan Avenue, Albany, NY 12210) Safety Profile t>
>Confirmed carcinogen with experimental tumorigenic data. Poison by intratracheal route. Human systemic effects by inhalation: cough, dyspnea. About twice as toxic as silica in causing silicosis. Mutation data reported.
What about recycling the crumbled and crushed cement? I personally wonder about dispersing more chemical particles into the air if they take this material and crush it to reform it into new materials. I would like to see some statistics about air quality and efficiency.
Once i got a homeopathic medicine by the name of "DUST" in Karachi,a company name Lemasol was producing it,I do not know if it was a local or a German company,in my family for dust allergy,and found it useful for acute prescription.My wife is dust allergic and has used it a couple of times.
I do not have any write up on it as such can not comment,but if any other HOMEOPATH has had experience with this medicine,he can share his experience with HWC,
As far as I can remember, there is drug called “House dust”. I had read about its provings in an old issue of B.H.J. After I found your post I am trying to search out the literature and will tell you if found.
I do not think "house dust" is the same thing. I believe that may be common dust in the interior of the home with such things as mites and animal fur. The dust we are seeking in terms of isopathy is the building materials, the walls and foundation with many chemicals.